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dc.contributor.authorWei, Qing
dc.contributor.authorTarighi, Saeed
dc.contributor.authorDötsch, Andreas
dc.contributor.authorHäussler, Susanne
dc.contributor.authorMüsken, Mathias
dc.contributor.authorWright, Victoria J
dc.contributor.authorCámara, Miguel
dc.contributor.authorWilliams, Paul
dc.contributor.authorHaenen, Steven
dc.contributor.authorBoerjan, Bart
dc.contributor.authorBogaerts, Annelies
dc.contributor.authorVierstraete, Evy
dc.contributor.authorVerleyen, Peter
dc.contributor.authorSchoofs, Liliane
dc.contributor.authorWillaert, Ronnie
dc.contributor.authorDe Groote, Valérie N
dc.contributor.authorMichiels, Jan
dc.contributor.authorVercammen, Ken
dc.contributor.authorCrabbé, Aurélie
dc.contributor.authorCornelis, Pierre
dc.date.accessioned2012-08-09T10:58:11Z
dc.date.available2012-08-09T10:58:11Z
dc.date.issued2011
dc.identifier.citationPhenotypic and genome-wide analysis of an antibiotic-resistant small colony variant (SCV) of Pseudomonas aeruginosa. 2011, 6 (12):e29276 PLoS ONEen_GB
dc.identifier.issn1932-6203
dc.identifier.pmid22195037
dc.identifier.doi10.1371/journal.pone.0029276
dc.identifier.urihttp://hdl.handle.net/10033/237935
dc.description.abstractSmall colony variants (SCVs) are slow-growing bacteria, which often show increased resistance to antibiotics and cause latent or recurrent infections. It is therefore important to understand the mechanisms at the basis of this phenotypic switch.
dc.language.isoenen
dc.rightsArchived with thanks to PloS oneen_GB
dc.titlePhenotypic and genome-wide analysis of an antibiotic-resistant small colony variant (SCV) of Pseudomonas aeruginosa.en
dc.typeArticleen
dc.contributor.departmentResearch Group Microbiology, VIB Department of Structural Biology, Vrije Universiteit Brussel, Brussels, Belgium.en_GB
dc.identifier.journalPloS oneen_GB
refterms.dateFOA2018-06-13T00:43:33Z
html.description.abstractSmall colony variants (SCVs) are slow-growing bacteria, which often show increased resistance to antibiotics and cause latent or recurrent infections. It is therefore important to understand the mechanisms at the basis of this phenotypic switch.


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