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dc.contributor.authorWilk, Esther
dc.contributor.authorPandey, Ashutosh K
dc.contributor.authorLeist, Sarah Rebecca
dc.contributor.authorHatesuer, Bastian
dc.contributor.authorPreusse, Matthias
dc.contributor.authorPommerenke, Claudia
dc.contributor.authorWang, Junxi
dc.contributor.authorSchughart, Klaus
dc.date.accessioned2015-09-24T15:12:18Zen
dc.date.available2015-09-24T15:12:18Zen
dc.date.issued2015en
dc.identifier.citationRNAseq expression analysis of resistant and susceptible mice after influenza A virus infection identifies novel genes associated with virus replication and important for host resistance to infection. 2015, 16:655 BMC Genomicsen
dc.identifier.issn1471-2164en
dc.identifier.pmid26329040en
dc.identifier.doi10.1186/s12864-015-1867-8en
dc.identifier.urihttp://hdl.handle.net/10033/578689en
dc.description.abstractThe host response to influenza A infections is strongly influenced by host genetic factors. Animal models of genetically diverse mouse strains are well suited to identify host genes involved in severe pathology, viral replication and immune responses. Here, we have utilized a dual RNAseq approach that allowed us to investigate both viral and host gene expression in the same individual mouse after H1N1 infection.
dc.language.isoenen
dc.titleRNAseq expression analysis of resistant and susceptible mice after influenza A virus infection identifies novel genes associated with virus replication and important for host resistance to infection.en
dc.typeArticleen
dc.contributor.departmentHelmholtz Centre for Infection research, Inhoffenstr. 7, D-38124 Braunschweig, Germany.en
dc.identifier.journalBMC genomicsen
refterms.dateFOA2018-06-13T04:08:35Z
html.description.abstractThe host response to influenza A infections is strongly influenced by host genetic factors. Animal models of genetically diverse mouse strains are well suited to identify host genes involved in severe pathology, viral replication and immune responses. Here, we have utilized a dual RNAseq approach that allowed us to investigate both viral and host gene expression in the same individual mouse after H1N1 infection.


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