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Authors
Stöhr, StefanieCosta, Rui
Sandmann, Lisa
Westhaus, Sandra
Pfaender, Stephanie
Anggakusuma
Dazert, Eva
Meuleman, Philip
Vondran, Florian W R
Manns, Michael P
Steinmann, Eike
von Hahn, Thomas
Ciesek, Sandra
Issue Date
2015-08-14
Metadata
Show full item recordAbstract
Chronically HCV-infected orthotopic liver transplantation (OLT) recipients appear to have improved outcomes when their immunosuppressive regimen includes a mammalian target of rapamycin (mTOR) inhibitor. The mechanism underlying this observation is unknown.Citation
Host cell mTORC1 is required for HCV RNA replication. 2015: GutAffiliation
TWINCORE, Centre for Experimental and Clinical Infection Research; a joint venture between the Medical School Hannover (MHH) and the Helmholtz Centre for Infection Research (HZI), Hannover, Germany.Journal
GutPubMed ID
26276683Type
ArticleISSN
1468-3288ae974a485f413a2113503eed53cd6c53
10.1136/gutjnl-2014-308971
Scopus Count
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