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dc.contributor.authorSchulz, Christian
dc.contributor.authorSchütte, Kerstin
dc.contributor.authorKoch, Nadine
dc.contributor.authorVilchez-Vargas, Ramiro
dc.contributor.authorWos-Oxley, Melissa L
dc.contributor.authorOxley, Andrew P A
dc.contributor.authorVital, Marius
dc.contributor.authorMalfertheiner, Peter
dc.contributor.authorPieper, Dietmar H
dc.date.accessioned2017-01-05T16:22:12Z
dc.date.available2017-01-05T16:22:12Z
dc.date.issued2016-12-05
dc.identifier.citationThe active bacterial assemblages of the upper GI tract in individuals with and without Helicobacter infection. 2016 Guten
dc.identifier.issn1468-3288
dc.identifier.pmid27920199
dc.identifier.doi10.1136/gutjnl-2016-312904
dc.identifier.urihttp://hdl.handle.net/10033/620680
dc.description.abstractPatients infected with Helicobacter pylori develop chronic gastritis with a subgroup progressing to further complications. The role of microbiota from the oral cavity swallowed with saliva and either transiting the stomach or persisting in the gastric mucosa is uncertain. It is also not known whether the bacterial community differs in luminal and mucosal niches. A key question is whether H. pylori influences the bacterial communities of gastroduodenal niches.
dc.language.isoenen
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/4.0/*
dc.titleThe active bacterial assemblages of the upper GI tract in individuals with and without Helicobacter infection.en
dc.typeArticleen
dc.contributor.departmentHel,holtz Centre for infection research, Inhoffenstr. 7, 38124 Braunschweig, Germany.en
dc.identifier.journalGuten
refterms.dateFOA2018-05-23T12:10:12Z
html.description.abstractPatients infected with Helicobacter pylori develop chronic gastritis with a subgroup progressing to further complications. The role of microbiota from the oral cavity swallowed with saliva and either transiting the stomach or persisting in the gastric mucosa is uncertain. It is also not known whether the bacterial community differs in luminal and mucosal niches. A key question is whether H. pylori influences the bacterial communities of gastroduodenal niches.


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