The active bacterial assemblages of the upper GI tract in individuals with and without Helicobacter infection.
dc.contributor.author | Schulz, Christian | |
dc.contributor.author | Schütte, Kerstin | |
dc.contributor.author | Koch, Nadine | |
dc.contributor.author | Vilchez-Vargas, Ramiro | |
dc.contributor.author | Wos-Oxley, Melissa L | |
dc.contributor.author | Oxley, Andrew P A | |
dc.contributor.author | Vital, Marius | |
dc.contributor.author | Malfertheiner, Peter | |
dc.contributor.author | Pieper, Dietmar H | |
dc.date.accessioned | 2017-01-05T16:22:12Z | |
dc.date.available | 2017-01-05T16:22:12Z | |
dc.date.issued | 2016-12-05 | |
dc.identifier.citation | The active bacterial assemblages of the upper GI tract in individuals with and without Helicobacter infection. 2016 Gut | en |
dc.identifier.issn | 1468-3288 | |
dc.identifier.pmid | 27920199 | |
dc.identifier.doi | 10.1136/gutjnl-2016-312904 | |
dc.identifier.uri | http://hdl.handle.net/10033/620680 | |
dc.description.abstract | Patients infected with Helicobacter pylori develop chronic gastritis with a subgroup progressing to further complications. The role of microbiota from the oral cavity swallowed with saliva and either transiting the stomach or persisting in the gastric mucosa is uncertain. It is also not known whether the bacterial community differs in luminal and mucosal niches. A key question is whether H. pylori influences the bacterial communities of gastroduodenal niches. | |
dc.language.iso | en | en |
dc.rights.uri | http://creativecommons.org/licenses/by-nc-sa/4.0/ | * |
dc.title | The active bacterial assemblages of the upper GI tract in individuals with and without Helicobacter infection. | en |
dc.type | Article | en |
dc.contributor.department | Hel,holtz Centre for infection research, Inhoffenstr. 7, 38124 Braunschweig, Germany. | en |
dc.identifier.journal | Gut | en |
refterms.dateFOA | 2018-05-23T12:10:12Z | |
html.description.abstract | Patients infected with Helicobacter pylori develop chronic gastritis with a subgroup progressing to further complications. The role of microbiota from the oral cavity swallowed with saliva and either transiting the stomach or persisting in the gastric mucosa is uncertain. It is also not known whether the bacterial community differs in luminal and mucosal niches. A key question is whether H. pylori influences the bacterial communities of gastroduodenal niches. |