Homologous recombination mediates stable Fah gene integration and phenotypic correction in tyrosinaemia mouse-model.
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Authors
Junge, NormanYuan, Qinggong
Vu, Thu Huong
Krooss, Simon
Bednarski, Christien
Balakrishnan, Asha
Cathomen, Toni
Manns, Michael P

Baumann, Ulrich
Sharma, Amar Deep
Ott, Michael
Issue Date
2018-02-27
Metadata
Show full item recordAbstract
To stably correct tyrosinaemia in proliferating livers of fumarylacetoacetate-hydrolase knockout (Fah-/-)mice by homologous-recombination-mediated targeted addition of theFahgene.Citation
Homologous recombination mediates stable Fah gene integration and phenotypic correction in tyrosinaemia mouse-model. 2018, 10 (2):277-286 World J HepatolAffiliation
TWINCORE, Zentrum für experimentelle und klinischeInfektionsforschung GmbH, Feodor-Lynen-Str. 7, 30625 Hannover, Germany.Journal
World journal of hepatologyPubMed ID
29527263Type
ArticleLanguage
enISSN
1948-5182ae974a485f413a2113503eed53cd6c53
10.4254/wjh.v10.i2.277
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- Creative Commons
Except where otherwise noted, this item's license is described as http://creativecommons.org/licenses/by-nc-sa/4.0/