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dc.contributor.authorJunge, Norman
dc.contributor.authorYuan, Qinggong
dc.contributor.authorVu, Thu Huong
dc.contributor.authorKrooss, Simon
dc.contributor.authorBednarski, Christien
dc.contributor.authorBalakrishnan, Asha
dc.contributor.authorCathomen, Toni
dc.contributor.authorManns, Michael P
dc.contributor.authorBaumann, Ulrich
dc.contributor.authorSharma, Amar Deep
dc.contributor.authorOtt, Michael
dc.date.accessioned2018-04-05T11:55:04Z
dc.date.available2018-04-05T11:55:04Z
dc.date.issued2018-02-27
dc.identifier.citationHomologous recombination mediates stable Fah gene integration and phenotypic correction in tyrosinaemia mouse-model. 2018, 10 (2):277-286 World J Hepatolen
dc.identifier.issn1948-5182
dc.identifier.pmid29527263
dc.identifier.doi10.4254/wjh.v10.i2.277
dc.identifier.urihttp://hdl.handle.net/10033/621336
dc.description.abstractTo stably correct tyrosinaemia in proliferating livers of fumarylacetoacetate-hydrolase knockout (Fah-/-)mice by homologous-recombination-mediated targeted addition of theFahgene.
dc.language.isoenen
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/4.0/*
dc.titleHomologous recombination mediates stable Fah gene integration and phenotypic correction in tyrosinaemia mouse-model.en
dc.typeArticleen
dc.contributor.departmentTWINCORE, Zentrum für experimentelle und klinischeInfektionsforschung GmbH, Feodor-Lynen-Str. 7, 30625 Hannover, Germany.en
dc.identifier.journalWorld journal of hepatologyen
refterms.dateFOA2018-05-23T10:55:13Z
html.description.abstractTo stably correct tyrosinaemia in proliferating livers of fumarylacetoacetate-hydrolase knockout (Fah-/-)mice by homologous-recombination-mediated targeted addition of theFahgene.


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