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dc.contributor.authorHerzmann, C.
dc.contributor.authorDallenga, T.
dc.contributor.authorKalinke, U.
dc.date.accessioned2018-11-13T14:18:26Z
dc.date.available2018-11-13T14:18:26Z
dc.identifier.issn0934-8387
dc.identifier.issn1438-8790
dc.identifier.doi10.1055/s-0043-122961
dc.identifier.urihttp://hdl.handle.net/10033/621561
dc.description.abstractTuberculosis is transmitted by inhalation of Mycobacterium tuberculosis-containing aerosols; 75 % of all patients show pulmonary manifestation. Immune responses after exposure that lead to clinical symptoms occur mainly in the respiratory tract and are only poorly understood. In most cases, cells of the innate immune system are believed to control the growth of or eradicate inhaled mycobacteria. However, this cannot be verified in vivo using standard methods. Subsequently, CD4+ and CD8+ T cell-driven adaptive immune responses are induced that attempt to control bacterial growth. The humoral defence appears to be less important. This article gives an overview of the current understanding of pulmonary immune mechanisms during exposure, latent infection, active disease and therapy of tuberculosis. Übersicht Herzmann C et al. Pulmonale Immunität bei … Pneumologie Hen_US
dc.relation.urlhttp://www.thieme-connect.de/DOI/DOI?10.1055/s-0043-122961en_US
dc.rightsAttribution-NonCommercial-ShareAlike 3.0 United States*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/3.0/us/*
dc.titlePulmonale Immunität bei Tuberkuloseen_US
dc.typeArticleen_US
dc.source.volume72
dc.source.issue07
dc.source.beginpage493
dc.source.endpage502
dc.source.journaltitlePneumologie


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