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dc.contributor.authorChenna Narendra, Sudeep
dc.contributor.authorChalise, Jaya Prakash
dc.contributor.authorBiggs, Sophie
dc.contributor.authorKalinke, Ulrich
dc.contributor.authorMagnusson, Mattias
dc.date.accessioned2018-11-28T13:30:01Z
dc.date.available2018-11-28T13:30:01Z
dc.date.issued2018-01-01
dc.identifier.issn1664-3224
dc.identifier.pmid29515584
dc.identifier.doi10.3389/fimmu.2018.00285
dc.identifier.urihttp://hdl.handle.net/10033/621593
dc.description.abstractCD4 Arthritis was triggered by intra-articular injection of methylated bovine serum albumin (mBSA) in wild-type mice, Foxp3DTReGFP Both control mice and mice devoid of IFNAR-signaling in T helper cells were protected from arthritis by IFN-α. Depletion of T By activating IDO during antigen sensitization, IFN-α activates Ten_US
dc.publisherFrontiersen_US
dc.rightsAttribution-NonCommercial-ShareAlike 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/4.0/*
dc.subjectexperimental arthritisen_US
dc.subjectindoleamine 2,3-dioxygenaseen_US
dc.subjectinterferon-alphaen_US
dc.subjectkynurenineen_US
dc.subjectregulatory T-cellsen_US
dc.titleRegulatory T-Cells Mediate IFN-α-Induced Resistance against Antigen-Induced Arthritis.en_US
dc.typeArticleen_US
dc.contributor.departmentTWINCORE, Zentrum für experimentelle und klinische Infektionsforschung GmbH,Feodor-Lynen Str. 7, 30625 Hannover, Germany.en_US
refterms.dateFOA2018-11-28T13:30:02Z
dc.source.journaltitleFrontiers in immunology


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