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dc.contributor.authorGrigoryan, Gayane
dc.contributor.authorLonnemann, Niklas
dc.contributor.authorKorte, Martin
dc.date.accessioned2019-03-19T08:55:28Z
dc.date.available2019-03-19T08:55:28Z
dc.date.issued2019-01-01
dc.identifier.citationNeural Plast. 2019 Jan 21;2019:3152129. doi: 10.1155/2019/3152129. eCollection 2019.en_US
dc.identifier.issn1687-5443
dc.identifier.pmid30804990
dc.identifier.doi10.1155/2019/3152129
dc.identifier.urihttp://hdl.handle.net/10033/621726
dc.description.abstractPrenatal stress (PS) has long-term sequelae for the morphological and functional status of the central nervous system of the progeny. A PS-induced proinflammatory status of the organism may result in an impairment of both hippocampal synaptic plasticity and hippocampus-dependent memory formation in adults. We addressed here the question of how PS-induced alterations in the immune response in young and old mice may contribute to changes in hippocampal function in aging. Immune stimulation (viaen_US
dc.language.isoenen_US
dc.publisherHindawien_US
dc.rightsAttribution-NonCommercial-ShareAlike 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-sa/4.0/*
dc.titleImmune Challenge Alters Reactivity of Hippocampal Noradrenergic System in Prenatally Stressed Aged Mice.en_US
dc.typeArticleen_US
dc.contributor.departmentHZI, Helmholtz Zentrum für Infektionsforschung GmbH, Inhoffenstr. 7, 38124 Braunschweig Germany.en_US
dc.identifier.journalNeural plasticityen_US
refterms.dateFOA2019-03-19T08:55:28Z
dc.source.journaltitleNeural plasticity


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