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dc.contributor.authorBrandt, Sabine
dc.contributor.authorBallhause, Tobias M
dc.contributor.authorBernhardt, Anja
dc.contributor.authorBecker, Annika
dc.contributor.authorSalaru, Delia
dc.contributor.authorLe-Deffge, Hien Minh
dc.contributor.authorFehr, Alexander
dc.contributor.authorFu, Yan
dc.contributor.authorPhilipsen, Lars
dc.contributor.authorDjudjaj, Sonja
dc.contributor.authorMüller, Andreas J
dc.contributor.authorKramann, Rafael
dc.contributor.authorIbrahim, Mahmoud
dc.contributor.authorGeffers, Robert
dc.contributor.authorSiebel, Chris
dc.contributor.authorIsermann, Berend
dc.contributor.authorHeidel, Florian H
dc.contributor.authorLindquist, Jonathan A
dc.contributor.authorMertens, Peter R
dc.date.accessioned2022-06-13T08:23:25Z
dc.date.available2022-06-13T08:23:25Z
dc.date.issued2020-08-28
dc.date.submitted2019-12-13
dc.identifier.pmid32859670
dc.identifier.doi10.1681/ASN.2019121289
dc.identifier.urihttp://hdl.handle.net/10033/623201
dc.description.abstractKidney injuries that result in chronic inflammation initiate crosstalk between stressed resident cells and infiltrating immune cells. In animal models, whole-body receptor Notch3 deficiency protects from leukocyte infiltration and organ fibrosis. However, the relative contribution of Notch3 expression in tissue versus infiltrating immune cells is unknown.en_US
dc.language.isoenen_US
dc.rightsAttribution 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/*
dc.subjectcell adhesionen_US
dc.subjectcell signalingen_US
dc.subjectchronic inflammationen_US
dc.subjectchronic kidney diseaseen_US
dc.subjectfibrosisen_US
dc.titleFibrosis and Immune Cell Infiltration Are Separate Events Regulated by Cell-Specific Receptor Notch3 Expression.en_US
dc.typeArticleen_US
dc.identifier.eissn1533-3450
dc.identifier.journalJournal of the American Society of Nephrology : JASNen_US
dc.source.volume31
dc.source.issue11
dc.source.beginpage2589
dc.source.endpage2608
refterms.dateFOA2022-06-13T08:23:26Z
dc.source.journaltitleJournal of the American Society of Nephrology : JASN
dc.source.countryUnited States


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Attribution 4.0 International
Except where otherwise noted, this item's license is described as Attribution 4.0 International