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dc.contributor.authorWells, Philippa M
dc.contributor.authorAdebayo, Adewale S
dc.contributor.authorBowyer, Ruth C E
dc.contributor.authorFreidin, Maxim B
dc.contributor.authorFinckh, Axel
dc.contributor.authorStrowig, Till
dc.contributor.authorLesker, Till Robin
dc.contributor.authorAlpizar-Rodriguez, Deshire
dc.contributor.authorGilbert, Benoit
dc.contributor.authorKirkham, Bruce
dc.contributor.authorCope, Andrew P
dc.contributor.authorSteves, Claire J
dc.contributor.authorWilliams, Frances M K
dc.date.accessioned2022-08-10T11:19:28Z
dc.date.available2022-08-10T11:19:28Z
dc.date.issued2020-06-25
dc.identifier.pmid33345197
dc.identifier.doi10.1016/S2665-9913(20)30064-3
dc.identifier.urihttp://hdl.handle.net/10033/623248
dc.description.abstractWe found that presence of Prevotella spp were positively associated with the rheumatoid arthritis PRS in TwinsUK participants (q<1 × 10-7). This finding was validated in SCREEN-RA participants (n=133) carrying established shared epitope risk alleles (q=0·0011). We also found an association between Prevotella spp and presence of preclinical rheumatoid arthritis phases (q=0·021).en_US
dc.language.isoenen_US
dc.rightsAttribution 4.0 International*
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/*
dc.titleAssociations between gut microbiota and genetic risk for rheumatoid arthritis in the absence of disease: a cross-sectional study.en_US
dc.typeArticleen_US
dc.identifier.eissn2665-9913
dc.identifier.journalThe Lancet. Rheumatologyen_US
dc.source.volume2
dc.source.issue7
dc.source.beginpagee418
dc.source.endpagee427
refterms.dateFOA2022-08-10T11:19:28Z
dc.source.journaltitleThe Lancet. Rheumatology
dc.source.countryUnited Kingdom
dc.source.countryUnited Kingdom
dc.source.countryUnited Kingdom
dc.source.countryEngland


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Attribution 4.0 International
Except where otherwise noted, this item's license is described as Attribution 4.0 International